NSAIDs are anti-beta amyloid
Although definitive evidence is lacking that NSAIDs can protect against the development of Alzheimer's disease, a new laboratory study has shown that they could reduce the characteristic deposition of beta amyloid protein in the brain - and ibuprofen may be among the most active (Neuropharmacology 2005;49:1088-99).

In vitro, NSAIDs inhibit the formation and extension of beta-amyloid fibrils from fresh protein, and they also destabilise preformed fibrils. The most active NSAIDs were ibuprofen, sulindac and mefenamic acid, followed by aspirin, other propionic acids, diclofenac and naproxen, with indomethacin having least activity.

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